Year : 2013  |  Volume : 12  |  Issue : 1  |  Page : 90-94

Chemical constituents from the aerial parts of Salsola inermis

1 Department of Chemistry of Natural and Microbial Products, National Research Center, Dokki, Egypt; Collage of Science and Humanities, Salman bin Abdul Aziz University, Alkharj City, Kingdom of Saudi Arabia
2 Department of Theraputic Chemistry, National Research Center, Dokki, Egypt

Correspondence Address:
Fatma S. Elsharabasy
PhD, Department of Chemistry of Natural and Microbial Products, National Research Center, El-Behooth St, Dokki 12311, Egypt

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Source of Support: None, Conflict of Interest: None

DOI: 10.7123/01.EPJ.0000428060.24957.95

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Background and objective

The hydroalcoholic extract from the aerial parts of Salsola inermis exhibited antioxidant, anti-inflammatory, and antinociceptive effects. The present study deals with the isolation and identification of the chemical constituents of this hydroalcoholic extract.

Materials and methods

The aerial parts of S. inermis (Forsskal) were collected from wild plants growing near the El-Alamein area in October 2005. Air-dried and powdered aerial parts of S. inermis were extracted with 70% alcohol in H2O. The extract was partitioned successively with CHCl3, EtOAc, and n-BuOH. The structures of the isolated compounds were determined by chemical and spectroscopic analyses.

Results and conclusion

Phytochemical investigation of the alcoholic extract from the aerial parts of S. inermis revealed 12 compounds, identified as long chain hydroxyl fatty acid 9,12,13-trihydroxydecosan–10,15,19-trienoic acid; trans-N-feruloyl tyramine-4ººº-O-β-D-glucopyranoside; umbelliferone; scopoletin; 3-methyl kaempferol; olean-12-en-3,28-diol; olean-12-en-28-oic acid; stigmasterol-3-β-O-D-glucopyranoside; 3-O-[β-D-glucopyranosyl]oleanolic acid; kaempferol 3-O-β-glucopyranoside; and isorhamnetin 3-O-β-glucopyranoside, in addition to β-sitosterol, stigmasterol, and stigmastanol.

Some of these compounds have hydroxyl groups, which help in scavenging free radicals and inhibit COX and various mediators involved in the pathogenesis of pain relief.

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